Xu Peng, Qiong Lin, Yang Liu, Yixin Jin, Joseph E. Druso, Marc A. Antonyak, Jun-Lin Guan, Richard A. Cerione. Inactivation of Cdc42 in embryonic brain results in hydrocephalus with ependymal cell defects in mice[J]. Protein&Cell, 2013, 4(8): 641-641. doi: 10.1007/s13238-013-0804-3
Citation:
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Xu Peng, Qiong Lin, Yang Liu, Yixin Jin, Joseph E. Druso, Marc A. Antonyak, Jun-Lin Guan, Richard A. Cerione. Inactivation of Cdc42 in embryonic brain results in hydrocephalus with ependymal cell defects in mice[J]. Protein&Cell, 2013, 4(8): 641-641. doi: 10.1007/s13238-013-0804-3
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Xu Peng, Qiong Lin, Yang Liu, Yixin Jin, Joseph E. Druso, Marc A. Antonyak, Jun-Lin Guan, Richard A. Cerione. Inactivation of Cdc42 in embryonic brain results in hydrocephalus with ependymal cell defects in mice[J]. Protein&Cell, 2013, 4(8): 641-641. doi: 10.1007/s13238-013-0804-3
Citation:
|
Xu Peng, Qiong Lin, Yang Liu, Yixin Jin, Joseph E. Druso, Marc A. Antonyak, Jun-Lin Guan, Richard A. Cerione. Inactivation of Cdc42 in embryonic brain results in hydrocephalus with ependymal cell defects in mice[J]. Protein&Cell, 2013, 4(8): 641-641. doi: 10.1007/s13238-013-0804-3
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Inactivation of Cdc42 in embryonic brain results in hydrocephalus with ependymal cell defects in mice
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1 Department of Systems Biology and Translational Medicine, College of Medicine, Texas A & M Health Science Center, Temple, TX 76504, USA;
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2 Department of Molecular Medicine, Cornell University, Ithaca, NY 14853, USA;
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3 Department of Internal Medicine-Division of Molecular Medicine and Genetics and Department of Cell and Developmental Biology, University of Michigan Medical School, Ann Arbor, MI 48109, USA